Search results for "Endogenous opioid"

showing 10 items of 21 documents

Lack of plasmic beta-endorphin response to a gastronomic meal in healthy humans.

1991

Abstract In order to study the relationship between the endogenous opiate system and food intake in man, plasma concentrations of beta-endorphin were measured in ten healthy subjects. Time course of beta-endorphinemia was compared under the following conditions: basal (fasting), after an injection of pentagastrin (6 μg/kg), or after a gastronomic meal. No changes in plasma beta-endorphin or ACTH concentrations were observed with pentagastrin nor after the meal, despite the combination of very high sensory pleasure with intake of a very large amount of food. It is concluded that blood beta-endorphin concentration is not a sensitive index of the effects of food intake on the endogenous opioid…

AdultBlood GlucoseMaleendocrine systemmedicine.medical_specialtyHungerAppetiteExperimental and Cognitive PsychologyPeptide hormoneBehavioral Neurosciencechemistry.chemical_compoundEatingInternal medicinemedicineHumansPalatabilityEndogenous opioidMealbusiness.industrydigestive oral and skin physiologybeta-EndorphinPentagastrinAffectEndocrinologychemistryBasal (medicine)TasteReceptors Opioidbeta-EndorphinOpiatebusinessArousalEnergy Intakehormones hormone substitutes and hormone antagonistsmedicine.drugPhysiologybehavior
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The Endogenous Opioid System Is Not Involved in Modulation of Opioid-Induced Hyperalgesia

2009

Abstract Some recent studies suggested a role of the endogenous opioid system in modulating opioid-induced hyperalgesia (OIH). In order to test this hypothesis, we conducted a prospective randomized, placebo-controlled, 2-way crossover study in healthy human volunteers. We utilized a well-established model of inducing OIH after a brief exposure to the μ-opioid agonist remifentanil using intradermal electrical stimulation. Patients were exposed to a randomized 90-minute infusion of remifentanil or saline placebo during 2 separate occasions. Development of OIH was quantified using changes in the average radius of the area of secondary hyperalgesia generated by electrical pain stimulation. A 2…

AdultMaleAgonistmedicine.drug_classNarcotic AntagonistsRemifentanilBlood PressureStimulationPharmacologyPlaceboRemifentanilYoung AdultDouble-Blind MethodPiperidinesHeart RatemedicineHumansOpioid-induced hyperalgesiaPain MeasurementEndogenous opioidCross-Over StudiesNaloxonebusiness.industryMiddle AgedAnalgesics OpioidAnesthesiology and Pain MedicineNeurologyOpioidHyperalgesiaAnesthesiaHyperalgesiaNeurology (clinical)medicine.symptombusinessmedicine.drugThe Journal of Pain
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Basal opioid receptor binding is associated with differences in sensory perception in healthy human subjects: a [18F]diprenorphine PET study.

2009

The endogenous opioid system is involved in many body functions including pain processing and analgesia. To determine the role of basal opioid receptor availability in the brain in pain perception, twenty-three healthy subjects underwent positron emission tomography (PET) utilizing the subtype-nonselective opioid antagonist [(18)F]diprenorphine, quantitative sensory testing (QST) and the cold pressor test. Binding potentials (BPs) were calculated using a non-invasive reference tissue model and statistical parametric mapping was applied for t-statistical analysis on a voxelwise basis. We found that cold pain-sensitive subjects present a significantly lower BP in regions including the bilater…

AdultMalePain Thresholdmedicine.drug_classCognitive NeuroscienceSensationDiprenorphinePainInsular cortexYoung AdultOpioid receptorOpioid Receptor BindingPhysical StimulationmedicinePressureHumansEndogenous opioidBrain ChemistryBrainSomatosensory CortexMiddle AgedCold TemperatureNeurologyOpioidData Interpretation StatisticalPositron-Emission TomographySensory ThresholdsReceptors OpioidOrbitofrontal cortexPerceptionRadiopharmaceuticalsPsychologyDiprenorphineNeuroscienceOpioid antagonistmedicine.drugNeuroImage
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Regional distribution of opioidergic nerves in human and canine prostates

1989

The regional distribution of opioidergic nerves in the juvenile and adult human prostate and in the adult canine prostate has been studied immunohistochemically using well-characterized polyclonal antisera against multiple opioid peptides. Nerves displaying immunoreactivity (ir) for the proenkephalin (PRO-ENK) derivatives met-enkephalin (ME), leuenkephalin (LE), octapeptide, and heptapeptide (ordered in decreasing frequency) were present in the dorsolateral stroma of human prostate. In canine prostate, the situation was similar, but the number of opioid-ir nerve fibers was lower than in human prostate. In both species, staining for the prodynorphin (PRO-DYN) derivatives dynorphin A and alph…

AdultMaleendocrine systemmedicine.medical_specialtyStromal cellAdolescentEnkephalin MethionineUrologyDynorphinBiologyEjaculatory ductchemistry.chemical_compoundDogsProstateInternal medicinemedicineAnimalsHumansOpioid peptideAgedEndogenous opioidProstateInfantDynorphin AMiddle AgedImmunohistochemistryPeptide FragmentsProenkephalinEndocrinologymedicine.anatomical_structureOncologychemistryChild PreschoolEnkephalin LeucineThe Prostate
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Endogenous opioid peptide responses to opioid and anti-inflammatory medications following eccentric exercise-induced muscle damage.

2009

To determine the effects of Vicoprofen, Ibuprofen, and a placebo on the responses of endogenous opioid peptides following eccentric exercise-induced muscle damage 36 healthy men (age: 22.8 years; height: 178.8+/-6.2cm; body mass: 78.9+/-13.7kg; body fat: 15.8+/-6.5%) volunteered to participate in the study. Each participant was evaluated for pain 24h post and randomly assigned to an experimental group: VIC (Vicoprofen), IBU (Ibuprofen), or P (placebo). Medication was given four times daily (i.e., VIC (hydrocodone bitartrate 7.5mg with Ibuprofen 200mg) and IBU 200mg). Blood was obtained at rest and at 0, 24, 48, 72, 96 and 120h following the eccentric exercise damage protocol. No significant…

AdultMalemedicine.medical_specialtyAdolescentPhysiologyPhysical ExertionAnti-Inflammatory AgentsPainPhysical exercisePlaceboBiochemistryPlacebosCellular and Molecular NeuroscienceYoung AdultEndocrinologyDouble-Blind MethodInternal medicinemedicineEccentricAnimalsHumansOpioid peptideMuscle SkeletalExerciseEndogenous opioidPain MeasurementChemistryIbuprofenMagnetic Resonance ImagingEndocrinologymedicine.anatomical_structureOpioidOpioid PeptidesAdrenal medullamedicine.drugPeptides
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Intraventricular insulin decreases kappa opioid-mediated sucrose intake in rats.

2002

The hormone insulin acts in the central nervous system (CNS) as a regulator of body adiposity and food intake. Recent work from our laboratory has provided evidence that one way by which insulin may decrease food intake is by decreasing the rewarding properties of food. Evidence from others suggests that endogenous opioids may mediate the palatable properties of foods, and insulin may decrease nonfood-related reward via interaction with some CNS kappa opioid systems. In the present study we examined the ability of insulin to interact with exogenous or endogenous kappa opioids to modulate feeding of palatable sucrose pellets by nondeprived rats. Insulin (5 mU intracerebroventricular (i.c.v.)…

AgonistMaleNarcoticsmedicine.medical_specialtySucrosePhysiologymedicine.drug_classmedicine.medical_treatmentNarcotic AntagonistsBiochemistryκ-opioid receptorNaltrexoneCellular and Molecular NeuroscienceEndocrinologyInternal medicinemedicineAnimalsInsulinEndogenous opioidInjections Intraventricularbusiness.industryInsulinReceptors Opioid kappa34-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide (trans)-IsomerFeeding BehaviorNaltrexoneRatsEndocrinologyOpioidbusinessNorbinaltorphiminemedicine.drugHormonePeptides
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On the opioid receptor subtype inhibiting the evoked release of 3H-noradrenaline from guinea-pig atria in vitro

1986

1. Guinea-pig isolated atria were incubated and loaded with 3H-(−)-noradrenaline. The intrinsic nerves were stimulated with trains of 5 or 35 field pulses (4 Hz), and the evoked efflux of 3H-noradrenaline and of total tritium was determined in the presence of atropine, corticosterone, desipramine, and phentolamine by liquid scintillation spectrometry. 2. Ethylketocyclazocine (1.4 nmol/l, IC50), MR 2033 (9.1 nmol/l), dynorphin A (1–13) (25 nmol/l, peptidase inhibitors present), etorphine (71 nmol/l), and [d-Ala2, d-Leu5]-enkephalin (>10 μmol/l, peptidase inhibitors present) inhibited the stimulation-evoked efflux of 3H-noradrenaline in a concentration-dependent manner, but not morphine up to…

Atropinemedicine.medical_specialtyEthylketocyclazocinemedicine.drug_classGuinea PigsPopulationEthylketocyclazocine(+)-NaloxoneIn Vitro TechniquesPharmacologyBinding CompetitiveDynorphinsNorepinephrinechemistry.chemical_compoundOpioid receptorInternal medicinemedicineAnimalsCyclazocineHeart AtriaPhentolamineeducationEndogenous opioidPharmacologyeducation.field_of_studyMorphineNaloxoneMyocardiumReceptors Opioid kappaDesipramineEtorphineDynorphin AGeneral MedicineEnkephalin Leucine-2-AlaninePeptide FragmentsBenzomorphansEndocrinologyEtorphineOpioidchemistryReceptors OpioidSynapsesCorticosteroneEnkephalin Leucinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Hunting for the high-affinity state of G-protein-coupled receptors with agonist tracers: Theoretical and practical considerations for positron emissi…

2019

Abstract The concept of the high‐affinity state postulates that a certain subset of G‐protein‐coupled receptors is primarily responsible for receptor signaling in the living brain. Assessing the abundance of this subset is thus potentially highly relevant for studies concerning the responses of neurotransmission to pharmacological or physiological stimuli and the dysregulation of neurotransmission in neurological or psychiatric disorders. The high‐affinity state is preferentially recognized by agonists in vitro. For this reason, agonist tracers have been developed as tools for the noninvasive imaging of the high‐affinity state with positron emission tomography (PET). This review provides an…

Central Nervous SystemBETA-ADRENERGIC-RECEPTORpositron emission tomographyagonist high-affinity stateD-2/3 AGONISTG-protein-coupled receptorsReview ArticleReceptors G-Protein-Coupledchemistry.chemical_compound0302 clinical medicineDrug DiscoveryReceptorNeurotransmitterReview Articles0303 health sciencesmedicine.diagnostic_testNONHUMAN PRIMATE BRAINEndocytosisTEST-RETEST REPRODUCIBILITYPositron emission tomographyG‐protein‐coupled receptors030220 oncology & carcinogenesisENDOGENOUS OPIOID RELEASEMolecular MedicineIN-VIVO BINDINGSignal TransductionAgonistNoninvasive imagingexperimental designmedicine.drug_classNeurotransmissionRAT-BRAINneurotransmittersagonist high‐affinity state03 medical and health sciencesIn vivomedicineAnimalsHumanshuman brain030304 developmental biologyG protein-coupled receptorPharmacologyDOPAMINE D2(HIGH) RECEPTORS5-HT1A RECEPTORSchemistryPositron-Emission TomographyPET RADIOLIGANDRadiopharmaceuticalsNeuroscienceMedicinal research reviews
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Role of Levo-tetrahydropalmatine and its metabolites for management of chronic pain and opioid use disorders.

2021

Abstract Background Opioids have been prescribed to reduce suffering from pain and to enhance quality of life. Due to the addictive potential and the lack of other effective alternatives to treat severe acute and chronic pains, opioids remain a serious public health issue. While, opioids directly influence the drug-seeking behavior, tolerance and withdrawal processes, through neuroadaptation, the brain's endogenous opioid system also adapts in the presence of chronic pain and could contribute to the difficulty of treatment. Despite the seemingly obvious interaction between the presence of pain and opioid-abuse, little is known about the underlying mechanisms in the brain. Purpose To review …

Drugmedia_common.quotation_subjectAnalgesicBerberine AlkaloidsPharmaceutical ScienceBioinformaticsPartial agonist03 medical and health sciences0302 clinical medicineDrug DiscoverymedicineHumans030304 developmental biologyEndogenous opioidmedia_commonPharmacology0303 health sciencesbusiness.industryAddictionChronic painOpioid use disordermedicine.diseaseOpioid-Related DisordersClinical trialAnalgesics OpioidComplementary and alternative medicine030220 oncology & carcinogenesisQuality of LifeMolecular MedicineChronic PainbusinessPhytomedicine : international journal of phytotherapy and phytopharmacology
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GHB differentially affects morphine actions on motor activity and social behaviours in male mice

2003

There are several reports suggesting that gamma-hydroxybutyric acid (GHB) influences the endogenous opioid system. The present study aimed to investigate the effects of GHB on motor and social activities and to examine its influence on morphine's actions on these behaviours. In a first experiment, several doses of GHB were studied but only the highest (200 and 400 mg/kg) produced a decrease in spontaneous motor activity measured in an actimeter cage. When hyperactivity induced by injecting 50 mg/kg of morphine was evaluated, all the GHB doses efficiently counteracted this morphine action. Using the paradigm of isolation-induced aggression, administration of 200 mg/kg of GHB significantly de…

MaleNarcoticsmedicine.drug_classClinical BiochemistryMale miceMotor ActivityPharmacologyToxicologyBiochemistryMiceBehavioral NeuroscienceOpioid receptormedicineAnimalsDrug InteractionsMotor activitySocial BehaviorBiological PsychiatryEndogenous opioidPharmacologyMorphineAggressionBiological activityAggressionOpioidExploratory BehaviorMorphinemedicine.symptomSodium OxybatePsychologyAnesthetics Intravenousmedicine.drugPharmacology Biochemistry and Behavior
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